Excess air with prolonged exposure can provoke the development of atherosclerosis, according to American scientists.

The inhalation of microparticles formed during fuel combustion enhances the processes of peroxidation in the body, causes inflammation in the walls of the arteries and gradually leads to the development of atherosclerotic vascular lesions, according to the Internet publication for girls and women from 14 to 35 years old Pannochka. Net This was discussed in the speech of Dr. Nino Kuenzil (University of Southern California) at the regular scientific session of the American Heart Association in New Orleans. Dr. Kuenzil and his colleagues analyzed the relationship between the carotid intima-media index (IMSA) and the level of air pollution in the area of ??residence, in particular the concentration of small particles, to 2. 5 microns in diameter (PM2. 5). It is these small particles that can penetrate into the most separated parts of the bronchial tree.

With the increase in PM2. 5 for every 10 mcg / cc. M, the IMHA index increased by 5. 9%. Even after adjusting for age, smoking status and various physiological parameters, the effect of pollutants remained statistically significant: with an increase in the PM2 concentration. 5 per 10 ?g / cc.

M, the ISMS index increased by 3. 9-4. 3%. The strongest was this relationship in women 60 years of age and older (IMSA increased by 15. 7%).

In the pathogenesis of this connection, both the autonomic nervous system and inflammation can participate, explains Dr. Kuenzil. Chronic subclinical inflammation develops, oxidized lipid fractions damage the vascular wall, it thickens, calcifies, plaques are formed, which eventually burst.

Medicus. En.

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