The AHA Nutrition Committee assumes that high protein intake can have a devastating effect on liver function. However, there is no scientific confirmation of this assumption. Protein is necessary not only for the restoration of liver tissue, but also for the formation of lipotropic agents: methionine and choline, which promote the conversion of fats to lipoproteins to remove them from the liver and thus prevent its fatty infiltration.

Rodents given large amounts of proteins demonstrated morphological changes in hepatic mitochondria, which may be pathological for a mopt. However, according to Jorda et al.. (Jorda et al), the liver responds to increasing protein in the diet by increasing the size of a normally functioning mitochondria. Moreover, the ratio of the number of amino acids with a branched chain to aromatic amino acids was also increased, indicating the absence of hepatic insufficiency in experimental animals. The authors concluded that "... increasing the protein content in the diet leads to a rapid increase in some indicators of hepatocytes ... The presented results are a clear example of how hepatocytes adapt to long-term metabolic stresses ...

" Further, protein catabolism in liver disease increases, and the lack of proteins in diet can exacerbate this process. With alcoholic cirrhosis of the liver, a high-calorie diet with a high protein content contributed to an improvement in liver function and reduced the risk of death. During one of the studies, this result was achieved by adding 60 g protein and 1,600 kcal during the first 30 days to the usual daily ration, followed by a decrease to 45 g protein and 1,200 kcal for the next 60 days.

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